Home Crop Science • Download Molecular Biology in Plant Pathogenesis and Disease by P. Narayanasamy PDF

Download Molecular Biology in Plant Pathogenesis and Disease by P. Narayanasamy PDF

By P. Narayanasamy

Reports at the phenomenon of plant pathogenesis (disease improvement) were helpful to have a deep perception into the interactions among host plant and the pathogen. reckoning on the degrees of susceptibility (compatibility) or resistance (incompatibility) of the host plant and virulence of the pathogen, ailment improvement might growth, both resulting in symptom expression or bring about the suppression of pathogen proliferation. Molecular thoughts were utilized to explain the character of interactions among the gene items of the plant and pathogen at mobile and molecular degrees. winning evasion of host s surveillance process and next actions of metabolites of the pathogen (enzymes and pollutants) encoded by way of pathogen genes counteracting the results of assorted defense-related antimicrobial compounds current already or produced by way of the host vegetation, after initiation of an infection were significantly studied by way of using a variety of molecular concepts. as well as learning numerous stages of affliction improvement in person vegetation, molecular tools were proven to be powerful, in amassing information on numerous features of epidemiology below ordinary stipulations the place the interplay of pathogen with populations of crops is stimulated considerably via the environmental stipulations present in several ecosystems. This quantity makes a speciality of the potential of utilizing the data on pathogenesis and molecular epidemiology to figure out the weak levels within the existence cycles of the pathogens that may be disrupted to accomplish more beneficial sickness regulate.

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Extra info for Molecular Biology in Plant Pathogenesis and Disease Management: Disease Development, Volume 2

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The EST set also provided the BcPLS1 gene, the homolog to MgPLS1 that is necessary for appressorium-mediated penetration of M. grisea into host plants. BcPLS1 and MgPLS1- null mutants had similar penetration defects (Viaud et al. 2005). Analysis of ESTs produced from the cDNA libraries of Magnaprothe grisea, representing a variety of growth conditions and cell types showed that about 23,000 of the ESTs could be clustered into 3050 contigs, leaving 5127 singleton sequences of the pathogen DNA. Analysis of ESTs provided the criteria for identification of fungal genes involved in pathogenesis.

In addition, Gna1 mutants did not sporulate and secreted one or more brown pigements into the growth medium. There was no such secretion from the wild-type. Gna1 appears to be the first signal transduction gene to be cloned and characterized from S. nodorum (Tan et al. 2004). For the fungal pathogens like Sclerotinia sclerotiorum, sclerotial development is a basic requirement for the progress of pathogenesis. A highly conserved homolog of ERK-type mitogen-activated protein kinases (MAPKs) from S.

ICL1 encodes isocitrate lyase involved in the glyoxylate cycle in peroxisomes. The icl1 mutants could not utilize fatty acids and acetate for growth. These mutants formed highly pigmented appressoria, suggesting that glyoxylate cycle was not required for melanin biosynthesis in appressoria. However, icl1 mutants had severely reduced virulence level and penetration hyphae failed to develop in the host plant, suggesting that ICL1 was involved in the invasion of host tissues. Partial restoration of virulence of icl1 mutant occurred on exogenous supply of glucose.

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