Home Nonfiction 10 • Download Arterial Lesions and Arteriosclerosis by H. Jellinek (auth.), H. Jellinek (eds.) PDF

Download Arterial Lesions and Arteriosclerosis by H. Jellinek (auth.), H. Jellinek (eds.) PDF

By H. Jellinek (auth.), H. Jellinek (eds.)

This monograph summarizes the result of a fifteen-year vascular pathology study venture subsidized via the Hungarian Academy of Sciences. the employees who've participated during this undertaking are indebted to the Academy for financing vast investigations and visits to institutes in another country, the goals of which have been to resolve definite difficulties via overseas cooperation. the implications awarded by way of the collective attempt of study groups, each one learning right here were completed a selected challenge, yet operating jointly to an identical finish. Mutual dialogue of findings and a standard alternate of knowledge among the study teams have contributed to the growth of the investigations. average ebook of info and mutual feedback ensured that the findings have been uniformly interpreted. it's was hoping that the elemental target of our long term undertaking, i. e. the choice of the perfect method of the complicated challenge of vascular pathology, has been completed. the recent process effects from the popularity that given that arteriosclerosis eventually results in different types of vascular damage, cognizance might be focussed at the occasions previous the improvement of the lesions instead of at the lesions themselves. This declare has been proven by means of a number of version experiments during which vascular damage inflicted less than various stipulations uniformly resulted in morphologi­ cally tested arteriosclerotic switch. the hot elevate within the prevalence of civi­ lization ailments of the vascular procedure has ended in a world-wide enlargement of arteriosclerosis research.

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Examinations have chiefly been carried out on newborn or embryonic rats, rabbit and chicks. Bartman (1968) studied the aortic ultrastructure of newborn rats, Bierring and Kabayashi (1963) that of newborn rabbits. Fahrenbach et al. (1965) examined the nuchal ligament of a calf foetus for elastic fibre structure) and described the elementary elastic unit of this system as a filament, 130 A in diameter. e. elastogenesis, is accompanied by a distinct cellular activity. Ross and Sandberg (1967) inferred from electron-microscopic examinations of embryonic rat tendon that one elementary unit of the elastic fibre is the so-called tubular fibril, 100 A in diameter, and another unit is the so-called central semi-amorphous substance.

Sensitivity to elastase is inversely related to the PI'A-affinity of the developing elastic tissue. Early elastic elements (granules, microfibrils, developing clumps and immature fibres) bind PI'A readily and resist the action of elastase, while mature elements (clumps, elastic fibres or elastic lamellae) gradually lose affinity to PI'A and simultaneously become susceptible to elastase. e. the ruthenium red-positive network of spheres and threads in the intercellular space of the chick embryo aorta appear in increasing amount from media to endothelium in the early stage of embryonic development between 6 and 8 days.

In the early stage, elastic clumps showing a marked affinity to PTA and uranyl acetate appeared below the endothelial cell layer and between the smooth muscle cells (Plates XIIj49, 50; XIII/53, 54). Elastic granules, fibrils and microfilaments were seen at the margins of the clumps and also independently of them (Plate XIIj47--49). The collagenous fibres appeared very early between the elastic ones, 37 and this was probably the only notable structural difference from the extracellular fibre genesis of the IP.

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